Terrance Kummer, MD, PhD, is an Associate Professor of Neurology at Washington University School of Medicine in St. Louis, a clinical neurointensivist in the Barnes-Jewish Hospital Neurological and Neurosurgical intensive Care Unit, and the Director of the Neurotrauma ICU. Dr. Kummer received his MD and PhD from Washington University in St. Louis where he uncovered a surprising muscle-dependent organizing influence on the complex, multiply-branched shape of the mature neuromuscular synapse. He completed his clinical training in Neurology and Neurocritical Care at Harvard Medical School (Massachusetts General Hospital and Brigham and Women’s Hospital). His work revealed that axonal injury is a component of hemorrhagic stroke, providing a pathophysiological link to traumatic brain injury (TBI). He now focuses on identifying and measuring the key loci of cellular injury in TBI in the hopes of uncovering its connection to Alzheimer’s disease, particularly in synaptic and other forms of CNS gray matter injury.
Collaborating PI
Terrance Kummer, MD, PhD
Washington University Medical Center
Terrance Kummer, MD, PhD, is an Associate Professor of Neurology at Washington University School of Medicine in St. Louis, a clinical neurointensivist in the Barnes-Jewish Hospital Neurological and Neurosurgical intensive Care Unit, and the Director of the Neurotrauma ICU. Dr. Kummer received his MD and PhD from Washington University in St. Louis where he uncovered a surprising muscle-dependent organizing influence on the complex, multiply-branched shape of the mature neuromuscular synapse. He completed his clinical training in Neurology and Neurocritical Care at Harvard Medical School (Massachusetts General Hospital and Brigham and Women’s Hospital). His work revealed that axonal injury is a component of hemorrhagic stroke, providing a pathophysiological link to traumatic brain injury (TBI). He now focuses on identifying and measuring the key loci of cellular injury in TBI in the hopes of uncovering its connection to Alzheimer’s disease, particularly in synaptic and other forms of CNS gray matter injury.
