Helen completed her PhD with Paul Greengard at The Rockefeller University where she generated genetic mouse models to study dopamine signaling and striatal-dependent behaviors. From there she joined Bernardo Sabatini’s lab as a post-doc at Harvard Medical School where she elucidated how mutations associated with neurodevelopmental disorders affect synaptic function and network activity. In 2013, Dr. Bateup started her lab at UC Berkeley in the Department of Molecular and Cell Biology. The goal of her research is to understand the cellular and molecular basis of neurodevelopmental and neurodegenerative disorders. Her work is defining how disease-associated genetic insults affect gene expression, signaling pathways, synaptic communication, and ultimately behavior.
Co-PI (Core Leadership)
Helen Bateup, PhD
University of California, Berkeley
Helen completed her PhD with Paul Greengard at The Rockefeller University where she generated genetic mouse models to study dopamine signaling and striatal-dependent behaviors. From there she joined Bernardo Sabatini’s lab as a post-doc at Harvard Medical School where she elucidated how mutations associated with neurodevelopmental disorders affect synaptic function and network activity. In 2013, Dr. Bateup started her lab at UC Berkeley in the Department of Molecular and Cell Biology. The goal of her research is to understand the cellular and molecular basis of neurodevelopmental and neurodegenerative disorders. Her work is defining how disease-associated genetic insults affect gene expression, signaling pathways, synaptic communication, and ultimately behavior.
