Sreeganga Chandra, PhD, received her undergraduate and master’s degrees in India. She earned a PhD in chemistry from Purdue University, and did her postdoctoral training with Dr. Edward Koo at UC San Diego and Dr. Thomas Südhof at University of Texas Southwestern Medical Center. In her postdoctoral work, she pursued her interests in neuroscience, specifically, the role of presynaptic proteins in neurodegeneration. Dr. Chandra started her career as a faculty member of the Yale School of Medicine. She is an associate professor in the Departments of Neurology and Neuroscience and is the deputy chair for the Department of Neuroscience at Yale. Dr. Chandra’s laboratory explores two inter-related themes–synapse maintenance and neurodegeneration, with a focus on Parkinson’s disease. Her lab studies familial Parkinson’s disease genes that encode synaptic proteins. They have extensively characterized the pathophysiology of alpha-synuclein, a key synaptic protein in the pathogenesis of Parkinson’s disease.
CO-INVESTIGATOR
Sreeganga Chandra, PhD
Yale University
Sreeganga Chandra, PhD, received her undergraduate and master’s degrees in India. She earned a PhD in chemistry from Purdue University, and did her postdoctoral training with Dr. Edward Koo at UC San Diego and Dr. Thomas Südhof at University of Texas Southwestern Medical Center. In her postdoctoral work, she pursued her interests in neuroscience, specifically, the role of presynaptic proteins in neurodegeneration. Dr. Chandra started her career as a faculty member of the Yale School of Medicine. She is an associate professor in the Departments of Neurology and Neuroscience and is the deputy chair for the Department of Neuroscience at Yale. Dr. Chandra’s laboratory explores two inter-related themes–synapse maintenance and neurodegeneration, with a focus on Parkinson’s disease. Her lab studies familial Parkinson’s disease genes that encode synaptic proteins. They have extensively characterized the pathophysiology of alpha-synuclein, a key synaptic protein in the pathogenesis of Parkinson’s disease.
