Bing Ren, PhD, is the Director of the Center for Epigenomics and Professor of Cellular and Molecular Medicine at the University of California at San Diego (UCSD). He is also a Member of the Ludwig Institute for Cancer Research (LICR). Dr. Ren obtained his Ph.D. in Biochemistry from Harvard University in 1998, and joined the faculty at LICR and UCSD in 2001, after completing postdoctoral training at the Whitehead Institute. Dr. Ren is studying how the non-coding sequences in the human genome direct spatiotemporal patterns of gene expression, how epigenetic mechanisms regulate their output during development, and how changes in these sequences contribute to human diseases. His lab has developed transformative tools for genome-wide analysis of enhancers and elucidated the chromatin features of these regulatory sequences. His pioneering work in epigenome analysis has laid the foundation for understanding how non-coding sequence variants contribute to complex traits and disease in humans.

University of California at San Diego | San Diego, USA

Bing Ren, PhD

University of California at San Diego | San Diego, USA

Bing Ren, PhD, is the Director of the Center for Epigenomics and Professor of Cellular and Molecular Medicine at the University of California at San Diego (UCSD). He is also a Member of the Ludwig Institute for Cancer Research (LICR). Dr. Ren obtained his Ph.D. in Biochemistry from Harvard University in 1998, and joined the faculty at LICR and UCSD in 2001, after completing postdoctoral training at the Whitehead Institute. Dr. Ren is studying how the non-coding sequences in the human genome direct spatiotemporal patterns of gene expression, how epigenetic mechanisms regulate their output during development, and how changes in these sequences contribute to human diseases. His lab has developed transformative tools for genome-wide analysis of enhancers and elucidated the chromatin features of these regulatory sequences. His pioneering work in epigenome analysis has laid the foundation for understanding how non-coding sequence variants contribute to complex traits and disease in humans.