Talia Lerner is an Assistant Professor of Neuroscience at Northwestern’s Feinberg School of Medicine. In her lab, she explores the brain circuitry underlying motivation and reinforcement learning, with a particular interest in how dopamine subcircuits coordinate to control learning trajectories and transitions between different strategies of reward-seeking. Dr. Lerner earned her BS in Molecular Biophysics & Biochemistry from Yale University. She earned her PhD in Neuroscience from UCSF under Dr. Anatol Kreitzer where she focused on understanding the molecular mechanisms of striatal synaptic plasticity, specifically on elucidating the roles of neuromodulators like dopamine in controlling the direction of plasticity. Dr. Lerner completed postdoctoral training at Stanford University under Dr. Karl Deisseroth, where she concentrated on the postsynaptic actions of dopamine to examine how dopamine release is regulated differentially in specific midbrain subcircuits.

Feinberg School of Medicine at Northwestern University | Chicago, USA
Co-Investigator

Talia Lerner, PhD

Feinberg School of Medicine at Northwestern University

Talia Lerner is an Assistant Professor of Neuroscience at Northwestern’s Feinberg School of Medicine. In her lab, she explores the brain circuitry underlying motivation and reinforcement learning, with a particular interest in how dopamine subcircuits coordinate to control learning trajectories and transitions between different strategies of reward-seeking. Dr. Lerner earned her BS in Molecular Biophysics & Biochemistry from Yale University. She earned her PhD in Neuroscience from UCSF under Dr. Anatol Kreitzer where she focused on understanding the molecular mechanisms of striatal synaptic plasticity, specifically on elucidating the roles of neuromodulators like dopamine in controlling the direction of plasticity. Dr. Lerner completed postdoctoral training at Stanford University under Dr. Karl Deisseroth, where she concentrated on the postsynaptic actions of dopamine to examine how dopamine release is regulated differentially in specific midbrain subcircuits.