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  • CamkIIa-cre;Ercc1-/fl mice

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    Neurons in the forebrain show elevated DNA damage and potential senescence, suggesting age-related cellular changes in the brain's higher cognitive functions.

  • Ercc1-/∆ mice

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    This research studies the model of a human progeroid syndrome

  • Nestin-cre;Ercc1-/fl mice

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    Pan-neuronal cell types may experience higher levels of internal DNA damage and potential senescence due to various factors.

  • Th-cre;Ercc1-/fl mice

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    Dopaminergic neurons may experience elevated DNA damage and potential senescence, suggesting implications for neurodegenerative disorders.

  • Oligo2-cre;Ercc1-/fl mice

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    Oligodendrocytes may experience higher levels of DNA damage and potential senescence due to internal factors.

  • Cx3cr1-cre;Ercc1-/fl mice

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    Microglia may experience higher levels of DNA damage and potential senescence due to internal factors.

  • Calbindin KD Mouse – Experimental Cross

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    Transgenic mouse model with decreased calb1 expression in DAT-expressing cells.

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